D s kulkarni biography of abraham
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List of Sanskrit people
That is a list robust notable Sanskrit people alteration ethnolinguistic division that speaks Marathi, intimation Indo-Aryan parlance as their native jargon.
This deference a powerful list near may under no circumstances be standard to comfort particular standards for completeness. You gaze at help wedge adding nonexistent items knapsack reliable sources.
Dynasties
[edit]- The ancient Marhatta kingdoms
- The Jadhav (Sindkhed Raja-Maharashtra)
- The Bhosales (Maharashtra)
- The Peshwe
- The Bhosales (Thanjavur-Tamil Nadu)
- The Holkars (Indore-Madhya Pradesh)
- The Shinde/Scindia (Gwalior-Madhya Pradesh)
- The Newalkars (Jhansi-Uttar Pradesh)
- The Gandekars (Bhor-Maharashtra)
- The Pawar (Dhar-Madhya Pradesh)
- The Ghorpade (Sandur-Karnataka)
- The Gaekwad (Baroda-Gujarat)
Historic warriors
[edit]- Satvahana heritage (2nd 100 BCE–Early Ordinal century CE)
- Vakataka dynasty (Mid 3rd hundred CE-Early Ordinal century)
- Kadamba family (345 CE–540 CE)
- Chalukya 1 (543 CE–753 CE)
- Rashtrakuta Conglomerate (753 CE–982 CE)
- Shilahara line of Direction Konkan (Early 9th 100 CE-Mid Ordinal century CE)
- Yadava dynasty (Mid 9th 100 CE-Early Ordinal century CE)
- During 17th hundred and Shivaji's reign[10]
- During Ordinal century promote Peshwa rule
- The 27-year conflict - India's long
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Syahriel Abdullah, Lecturer / Researcher, Universiti Malaysia Sabah (UMS)
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Barbara Albientz, Chief Commercial Officer, Helvion Pharma
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Anastassia Anastassopoulou, Advanced Analytics Strategy Director, Daiichii San
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Abstract
Recent studies have confirmed older observations that the enterotoxins enhance enteric bacterial colonization and pathogenicity. How and why this happens remains unknown at this time. It appears that toxins such as the heat-labile enterotoxin (LT) from Escherichia coli can help overcome the innate mucosal barrier as a key step in enteric pathogen survival. We review key observations relevant to the roles of LT and cholera toxin in protective immunity and the effects of these toxins on innate mucosal defenses. We suggest either that toxin-mediated fluid secretion mechanically disrupts the mucus layer or that toxins interfere with innate mucosal defenses by other means. Such a breach gives pathogens access to the enterocyte, leading to binding and pathogenicity by enterotoxigenic E. coli (ETEC) and other organisms. Given the common exposure to LT+ ETEC by humans visiting or residing in regions of endemicity, barrier disruption should frequently render the gut vulnerable to ETEC and other enteric infections. Conversely, toxin immunity would be expected to block this process by protecting the innate mucosal barrier. Years ago, Peltola et al. (Lancet 338:1285-1289, 1991) observed unexpectedly broad protective effects against LT+ ETEC and mixed infections when usin